Part,  Chapter, Paragraph

 1    I,     2.  8    |           burdens from occupational exposures~ ~Forms greenhouse gases,
 2    I,     2.  8    |           burdens from occupational exposures~ ~Forms greenhouse gases,
 3   II,     5.  2.  3|          changes in the environment exposures, health behaviours, diagnosis
 4   II,     5.  5.  3|        differences in environmental exposures, and/or different genetic
 5   II,     5.  5.  3|          effect, or primed by early exposures and with reciprocal interaction(
 6   II,     5.  5.  3|            studies of environmental exposures in Parkinson's disease.
 7   II,     5.  8.  4|           Survey (ECRHS) study high exposures to dusts and fumes increased
 8   II,     5.  8.  7|        Bronchitis, and Occupational Exposures in Young Adults Am. J. Respir.
 9   II,     5.  9.  4|          asthma due to occupational exposures ranged from 10% to 25%,
10   II,     5.  9.  4|            adults with occupational exposures. The relationship between
11   II,     5.  9.  4|   attributable to allergen or other exposures associated to pet ownership (
12   II,     5.  9.  4|          cities. It aims to measure exposures, collect information on
13   II,     5. 11.  5|           to decrease environmental exposures and give priority to preventing
14   II,     5. 11.  5|       legislation to reduce harmful exposures, may play an important part
15   II,     9        |     susceptibility to environmental exposures is likely to vary importantly
16   II,     9.  1.  2|             protection from adverse exposures to the entire community,
17   II,     9.  1.  2|           and agricultural chemical exposures (Bianchi et al, 2006).~ ~
18   II,     9.  1.  2|     susceptibility to environmental exposures is likely to vary importantly
19   II,     9.  1.  2|         drug exposure, occupational exposures and environmental pollution.
20   II,     9.  1.  2|            on rare anomalies and/or exposures, compare data between regions
21   II,     9.  1.  2| particularly with regard to complex exposures such as environmental pollution.~ ~
22  III,    10.  1    |        means that the links between exposures and their health consequences
23  III,    10.  1    |       burden of chemicals, combined exposures from multiple sources (food,
24  III,    10.  1    |  cocktail-effect' and the timing of exposures. The topic of 'Late responses
25  III,    10.  1    |         weather conditions~Multiple exposures~Leisure areas~Lifestyles~ ~ ~
26  III,    10.  1    |           most of the environmental exposures, physical factors such as
27  III,    10.  1    |       health in many ways – through exposures to physical, chemical and
28  III,    10.  1    |     associations with environmental exposures. The strengths of such associations
29  III,    10.  1    |         vary for different diseases/exposures – from a very likely association
30  III,    10.  1    |     associations with environmental exposures to chemicals and other environmental
31  III,    10.  1    |             with some environmental exposures~Infectious diseases~water,
32  III,    10.  1    |       sensitivity~multiple chemical exposures at low doses~ ~Note: many
33  III,    10.  2.  5|        traced back to environmental exposures early in life. Unfortunately
34  III,    10.  2.  5|       hinting at that environmental exposures early in life, probably
35  III,    10.  3.  1|        frequent, intermittent, high exposures to UVR (for example during
36  III,    10.  3.  1|           UVR in recent years. Peak exposures will probably occur around
37  III,    10.  3.  1|              seems to be limited to exposures above 0.4 µT. In European
38  III,    10.  3.  1|        twice as many workers report exposures to vibrations and noise
39  III,    10.  3.  1|              Most common in men are exposures to vibrations and noise
40  III,    10.  3.  1|            one quarter of the time. Exposures to inconvenient temperatures
41  III,    10.  3.  1|             are also rather common. Exposures in general are less often
42  III,    10.  3.  2|           appearing, resulting from exposures to low levels of an increasing
43  III,    10.  3.  2|             human and environmental exposures is incomplete. Increased
44  III,    10.  3.  2|     associations with environmental exposures to chemicals and other environmental
45  III,    10.  3.  2|             with some environmental exposures~Infectious diseases~water,
46  III,    10.  3.  2|       sensitivity~multiple chemical exposures at low doses~Source: European
47  III,    10.  3.  2|        traced back to environmental exposures early in life. Unfortunately,
48  III,    10.  3.  2|          hinting that environmental exposures early in life, probably
49  III,    10.  3.  2|            seafood); additional low exposures may come from consumer products
50  III,    10.  3.  4|             causes.~ ~Other harmful exposures were also caused or exacerbated
51  III,    10.  4.  1|      available for short-term acute exposures, there is little data about
52  III,    10.  4.  1|             the effect of long-term exposures. In a study made in the
53  III,    10.  4.  1|           characterised by combined exposures, where also the importance
54  III,    10.  4.  2|            for assessing the actual exposures of European consumes Article
55  III,    10.  4.  4|             consumer products~ ~ ~ ~Exposures to non-food consumer products,
56  III,    10.  4.  4|         products. For instance, low exposures to tin compounds, substances
57  III,    10.  4.  5|           and compare waste-related exposures with those due to other
58  III,    10.  4.  5|       caused by long term low-level exposures. In addition, the new generation
59  III,    10.  4.  5|           and compare wasterelated exposures with those due to other
60  III,    10.  4.  5|            metabolism and timing of exposures, and the analysis of all
61  III,    10.  5.  1|           lead to the initiation of exposures: cooking and showering increases
62  III,    10.  5.  1|         most of the health-relevant exposures of residential behaviour
63  III,    10.  5.  1|          poor ventilation, chemical exposures such as formaldehyde, moulds
64  III,    10.  5.  2|            more about environmental exposures and lack of amenities. Unpublished
65  III,    10.  5.  3|     compensation is still caused by exposures to silica even though the
66  III,    10.  5.  3|           compensation is caused by exposures to asbestos dust. This occupational
67  III,    10.  5.  3|          factors (table 10.5.3.13). Exposures to vibrations and noise -
68  III,    10.  5.  3|             are most common in men. Exposures to inconvenient temperatures
69  III,    10.  5.  3|             are also rather common. Exposures in general are less often
70  III,    10.  5.  3|        twice as many workers report exposures to vibrations and noise
71  III,    10.  6.  1|             2005).~ ~Figure 10.6.5. Exposures to assaults and threats
72   IV,    12.  2    |           lifestyles, environmental exposures to carcinogenic chemicals
73   IV,    13.  2.  2|              the health loss due to exposures from the physical environment
74  Key,   Ap5.  0.  0|   expenditure~expenditures~exposure~exposures~extreme~eye~eyecare~eyes~