Part,  Chapter, Paragraph

 1   II,     5.  2.  2|        the treatments. As already explained, MONICA data, although collected
 2   II,     5.  2.  3|     deaths than breast cancer.~As explained in chapter 5.2.2Data sources’,
 3   II,     5.  2.  3|    different populations could be explained by changes in the average
 4   II,     5.  2.  3|           above the age of 75. As explained in chapter 4.2.2Data sources’,
 5   II,     5.  2.  3|     east-West gap might be mostly explained by increased levels of traditional
 6   II,     5.  2.  4|           in classic risk factors explained only a part of the change
 7   II,     5.  2.  5|    including secondary prevention explained only the remaining 42% of
 8   II,     5.  5.  3|      among study estimates can be explained by the age of the children
 9   II,     5.  5.  3|         of reports can be largely explained by suboptimal case ascertainment
10   II,     5.  5.  3|           and women can be mostly explained by the differing genetic
11   II,     5.  5.  3|        The differences are mostly explained by the structure of the
12   II,     5.  5.  3|        French study can be mostly explained through accuracy in case
13   II,     5.  5.  3|           seizures may be largely explained by the different distribution
14   II,     5.  5.  3|          studies may be partially explained by the inclusion of acute
15   II,     5.  5.  3|           in children may be thus explained by the rate expected in
16   II,     5.  5.  3| difference in rates may be mostly explained by the small size of the
17   II,     5.  5.  3|    prevalence rate since 1978 was explained by the changing population
18   II,     5.  6.  3|         al, 1997) (Figure 5.6.1), explained partly by a cumulative effect
19   II,     5.  8.  3|         osteoporosis could not be explained by gluco-corticoid use alone,
20   II,     5.  9. FB|         Europe could be partially explained by the higher physicians’
21   II,     5.  9. FB|    allergies. This correlation is explained by the shift from Th-1 phenotype (
22   II,     5.  9.  3|         of centre level variation explained), hay fever (61 and 73%)
23   II,     5. 11.  3|       increases could probably be explained by increased cumulative
24   II,     9.  1.  1|       than neonatal mortality, as explained in Table 9.1.1.1. In addition,
25   II,     9.  3.  2|         deterioration can also be explained by increased risk factors
26  III,    10.  1.  1|        Conflicting results may be explained by the fact that the food
27  III,    10.  5.  3|    something that cannot be fully explained by the ‘healthy worker effect’,
28   IV,    11.  5.  4|      differences cannot be easily explained. They are probably due to
29   IV,    11.  5.  4|         understand. They could be explained by the wide variability
30   IV,    11.  5.  5|      access that cannot merely be explained by differences in donation
31   IV,    13.  2.  1|        more diseases which can be explained by that risk factor (or